Genetic analysis of BRCA1 function in a defined tumor cell line (Q28142643)

2

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K Vlasakova

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J Chen

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M Socolovsky

5

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D M Livingston

describes a project that uses

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language of work or name

English

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publication date

December 1999

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published in

Molecular Cell

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volume

4

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issue

6

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page(s)

1093-9

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HCC1937

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cites work

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Transcriptional activation by BRCA1

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Stable interaction between the products of the BRCA1 and BRCA2 tumor suppressor genes in mitotic and meiotic cells

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Requirement of ATM-dependent phosphorylation of brca1 in the DNA damage response to double-strand breaks

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Radiation-induced DNA double-strand breaks and the radiosensitivity of human cells: A closer look

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BRCA1 Required for Transcription-Coupled Repair of Oxidative DNA Damage

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Induction of GADD45 and JNK/SAPK-dependent apoptosis following inducible expression of BRCA1.

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SYBR Green I staining of pulsed field agarose gels is a sensitive and inexpensive way of quantitating DNA double-strand breaks in mammalian cells

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Differential induction of transcriptionally active p53 following UV or ionizing radiation: defects in chromosome instability syndromes?

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Targeted mutations of breast cancer susceptibility gene homologs in mice: lethal phenotypes of Brca1, Brca2, Brca1/Brca2, Brca1/p53, and Brca2/p53 nullizygous embryos

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Brca1 controls homology-directed DNA repair

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Structural changes in chromatin as the basis for radiosensitivity in ataxia telangiectasia

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Involvement of Brca2 in DNA Repair

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Location of BRCA1 in human breast and ovarian cancer cells

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Association of BRCA1 with Rad51 in mitotic and meiotic cells

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BRCA1 is a component of the RNA polymerase II holoenzyme

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Dynamic Changes of BRCA1 Subnuclear Location and Phosphorylation State Are Initiated by DNA Damage

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A targeted disruption of the murine Brca1 gene causes gamma-irradiation hypersensitivity and genetic instability.

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Update on breast cancer susceptibility genes

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Differential subcellular localization, expression and biological toxicity of BRCA1 and the splice variant BRCA1-delta11b

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Identification of a RING protein that can interact in vivo with the BRCA1 gene product

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Centrosome amplification and a defective G2-M cell cycle checkpoint induce genetic instability in BRCA1 exon 11 isoform-deficient cells

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Conditional mutation of Brca1 in mammary epithelial cells results in blunted ductal morphogenesis and tumour formation.

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The C-terminal (BRCT) domains of BRCA1 interact in vivo with CtIP, a protein implicated in the CtBP pathway of transcriptional repression

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

10635334

1 reference

Consolidated OpenCitations Corpus – April 2017