Protective effect of a 21-aminosteroid on the blood-brain barrier following subarachnoid hemorrhage in rats

Stroke. 1989 Mar;20(3):367-71. doi: 10.1161/01.str.20.3.367.

Abstract

The effects of subarachnoid injection of blood on blood-brain barrier permeability to albumin was assessed in a rat model. Subarachnoid injection of blood caused a significant sixfold increase in Evans blue extravasation, whereas sham operation or NaCl injection had no effect. In addition, subarachnoid injections of arachidonic acid or FeCl2 increased blood-brain barrier permeability to Evans blue 16- and 10-fold, respectively. The capillary permeability after subarachnoid injection of blood was normalized by pretreatment with a novel 21-aminosteroid, U-74006F, that has antioxidant and antilipolytic activity. Pretreatment with U-74006F also reduced the vascular leakage induced by subarachnoid injection of arachidonic acid or FeCl2 by 50% and 45%, respectively. We conclude that damage to membrane lipids by peroxidative and/or lipolytic processes is involved in the subarachnoid hemorrhage-induced blood-brain barrier opening and that U-74006F protects the blood-brain barrier against the effects of subarachnoid hemorrhage by preventing or limiting these pathologic membrane lipid changes.

MeSH terms

  • Animals
  • Arachidonic Acid
  • Arachidonic Acids / pharmacology
  • Blood-Brain Barrier / drug effects*
  • Brain / metabolism
  • Capillary Permeability / drug effects
  • Evans Blue
  • Extravasation of Diagnostic and Therapeutic Materials / metabolism
  • Ferrous Compounds / pharmacology
  • Male
  • Pregnatrienes / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Serum Albumin / metabolism
  • Subarachnoid Hemorrhage / chemically induced
  • Subarachnoid Hemorrhage / physiopathology*

Substances

  • Arachidonic Acids
  • Ferrous Compounds
  • Pregnatrienes
  • Serum Albumin
  • Arachidonic Acid
  • Evans Blue
  • ferrous chloride
  • tirilazad