Recent research has shown that some cells in recently dead organisms may be more genetically active than those in living ones. 😳 Learn more about the "twilight of life" in our study of the day:
John Templeton Foundation’s Post
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We are the only place in the world that possesses 18,000 gene clones out of the 20,000 human genomes that make up our DNA. With these clones, DNASU can conduct in-depth research of genetic mutations, diseases, and human health, better than any other lab on Earth. #plasmids #DNA #human #genes #DNASU #plasmidrepository #genes, #genome #clone #science #research
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Did you know that Carnitine Transport Deficiency (CTD) is significantly more prevalent in the Faroe Islands than anywhere else in the world? This hereditary condition, linked to mutations in the SLC22A5 gene, has raised alarms due to unexpected fatalities among young adults in this close-knit community. As researchers delve deeper into the genetic landscape, they are uncovering a complex web of factors that contribute to this phenomenon—ranging from historical population dynamics to geographical isolation. With over 140 diagnosed cases, the urgent push for screening and awareness continues to grow. The proactive measures taken by Faroese health authorities, including neonatal screening initiatives, highlight the importance of early detection and treatment. #CarnitineTransportDeficiency #GeneticResearch #HealthcareInnovation #FaroeIslands #Genetics #PublicHealth
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https://lnkd.in/eSM5Qiqa Article title: Transposable Elements in Fungi: A Genomic Approach Author(s): Mateus F. Santana; Marisa V. Queiroz Journal: Scientific Journal of Genetics and Gene Therapy Journal ISSN: 2640-7744 Abstract: Transposable elements (TEs) include a wide range of DNA sequences that can change positions in the genome. The accessibility of whole fungal genome sequences and analysis of TEs demonstrate the important role they play in genome evolution of fungi species. TE activity is a primary mechanism for high fitness, plasticity and adaptability in certain species of pathogenic fungi. Some of the deleterious effects of transposons may be due to ectopic recombination among transposons of the same family. To prevent possible damage caused by the transposons, some fungi possess TE-silencing mechanisms, for instance, RIP (Repeat Induced Pont mutation) and RNA silencing. In addition, TEs are efficient molecular markers due to their structure and transposition strategy. #Transposableelements #Pathogenicfungi #Repeatinducedpointmutation #Molecularmarkers #Genome #ClassicalAndDevelopmentalGenetics #ConservationAndEcologicalGenetics #EvolutionaryAndPopulationGenetics #GeneticEngineering #GeneticsOfPlantsAndAnimalsInAgronomy #Genomics #Peertechz #PeertechzPublications #HumanAndMedicalGenetics #QuantitativeGenetics #PopulationGenetics #PsychiatricGenetics #MicrobialGenetics #MolecularGenetics #GeneCharacterization #Polymorphisms #ControlOfGeneExpression #Epigenetics #TechnicalTipsAndImprovement #EpigeneticsAndChromosomeBiology #FunctionalGenetics #HumanPopulationGenetics #PlantPopulationGenetics #AnimalPopulationGenetics #ComplexTraits #QuantitativeGenetics #StatisticalAndComputationalGenetics #SomaticGeneTherapy #VectorsInGeneTherapy #Viruses #GermlineGeneTherapy #MajorDevelopmentsInGeneTherapy #NonViralMethods #PreventiveGeneTherapy
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Country Director & GM @Haier Biomedical | Board Member l Storyteller both by profession & passion | Believer in the India Story | Relentless Explorer |
The quest for a longer life is woven through human history. The researchers at the MRC Laboratory of Medical Science, Imperial College London and Duke-NUS Medical School in Singapore were investigating a protein called interleukin-11 & performed two experiments :- 1. The first genetically engineered mice so they were unable to produce interleukin-11 2. The second waited until mice were 75 weeks old (roughly equivalent to a 55-year-old person) and then regularly gave them a drug to purge interleukin-11 from their bodies A drug has increased the lifespans of laboratory animals by nearly 25%, in a discovery scientists hope can slow human ageing too. The results, published in the journal Nature, showed lifespans were increased by 20-25% depending on the experiment and sex of the mice. #AntiAgeing #LongLife #Science #Innovation
We'd all love to put the ageing process on ice. Luckily, scientists at MRC Laboratory of Medical Sciences (LMS) Imperial College London and Duke-NUS Medical School are making strides in slowing down ageing by targeting interleukin-11, a protein linked to inflammation and old age. Their findings are groundbreaking: genetically modified and drug-treated mice – dubbed 'supermodel grannies' – enjoy a 20-25 % increase in lifespan! It’s like freezing ageing in its tracks. Could this be the breakthrough to keep us 'forever young'? Read the full BBC News article to find out: https://ow.ly/Bc2N50TcAz4 #AgeingProcess #AntiAgeing #LongevityResearch #ForeverYoung #ScienceBreakthrough
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Happy to share our lab's first story about the function of Structural Maintenance of Chromosomes Complexes in nematodes, published today in Nature Genetics. Our team, led by (now) Dr. Moushumi Das, uncovered how Condensin I plays a crucial role in organizing the Caenorhabditis elegans genome during interphase. This discovery not only highlights the unique genome folding mechanisms in nematode holocentric chromosomes but also reveals significant impacts on X-chromosome gene regulation. Dive into the full paper to explore these function of these fascinating micromachines on chromosomal biology! https://rdcu.be/dOxQs Special thanks to our collaborators Collin Ewald (ETHZ) and Julien Mozziconacci (MNHN) #Genomics #ChromosomeBiology #Caenorhabditiselegans #NatureGenetics
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New research reveals that humans and other apes have a unique DNA insertion in the TBXT gene that is absent in primates with tails, such as monkeys. This genetic alteration also causes mice to have short or no tails. While this gene change contributes to tail loss, it's not the only factor. According to human geneticist Malte Spielmann, apes aren't the only primates without tails, indicating that the trait evolved independently multiple times. Co-author Bo Xia suggests that there are likely several ways to lose a tail during development, and our ancestors chose this particular path. Read more about this fascinating discovery in the link below. Source: https://lnkd.in/d_VynQb9
How humans lost their tails — and why the discovery took 2.5 years to publish
nature.com
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We've got another set of bite-sized science videos for you! In the sixth section of Science Live we are exploring how Duchenne muscular dystrophy is inherited and passed on through the generations. To do so we will explore topics on mutations and genetic inheritance, as well as looking at who carriers are and how the condition affects them. Genetically inherited conditions are complicated and often bring up complex emotions, and with this section of bite-sized videos we hope to provide you with the foundations to understand the inheritance of Duchenne. These videos are a great way to explain the complicated science behind Duchenne to friends, family and care teams. Always remember that we are here for you and your family wherever you are in your Duchenne journey - if you need support please get in touch: [email protected] https://lnkd.in/e5hjJ_5J #duchennescience #scienceeducation #understanding #knowledge #genetics #carriers #foreveryone
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🌟 A Revolutionary Twist in Early Embryo DNA Replication! 🧬 Forget what we thought we knew! Groundbreaking research reveals that DNA replication in early embryos is more unpredictable and dynamic than expected. This discovery could redefine our understanding of genetic stability and developmental biology as we know it. 🚀 Science never ceases to amaze, constantly pushing boundaries and reshaping what’s possible. Could this lead to new insights in gene regulation and embryonic development? What recent discoveries have made you rethink the fundamentals? 🤔 #Biology #Genetics #Research #DNA #Embryo #Innovation #ScienceBreakthroughs #STEM https://lnkd.in/exBYB3hk
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How migration affect allele frequencies Migration makes a groups within a population reproductively isolated and so gene flow between them stops since migration brings a group of them or both in another region environmental conditions change and each group adapt to new condition changing allelic frequency and the best fitted individuals pass on alleles to offspring and so two sub populations are formed and when they meet again they won't breed together but maybe Competition occur or one group is best fitted and the other whole group dies. isolation bring about the same results as about if any reproductive isolation occur, gene flow stops and gene pool is not maintained if no selection occur all individuals survive and since individuals reproduce sexually therefore variation occur due to random assortment of chromosomes in metaphase 1 and their consequent random distribution if them in the gamete cells. allelic frequency will then increase a lot since all individuals survive and are capable to reproduce and bring about new variation of offspring no random mating is like inbreeding in artificial selection where only closely relatives are allowed to breed in order to maintain the gene pool. Allelic frequency are then too minor only considering a double recessive character the express itself. very small population is taken as above where allelic frequency is too minor mutations are really different may happen is a sequence on the DNA or in whole set of chromosome or brings about polyploidy organisms. allelic frequency will absolutely vary if the new mutant organism id capable to survive but if not allelic frequency won't change Youtube video: https://lnkd.in/duqMXWV4 #nikolaysgeneticslessons
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https://lnkd.in/eNSaKbNU Article title: Voluntary Selection; Bringing Evolution at the Service of Humanity Author(s): Omid Sadeghi Fathabadi Journal: Scientific Journal of Genetics and Gene Therapy Journal ISSN: 2640-7744 Abstract: Human populations can be characterized by their corresponding phenotypic profiles which do not necessarily align. These differences are not our points of strength in building harmonious societies and establishing a sustainable world order; it is therefore desirable to seek a pathway to resolve them. This article reflects on genetics as the most profound source of our differences and introduces Voluntary Selection as a means of acquiring genetic qualities and resolving the imbalance in their possession by different populations. Voluntary Selection is a healthcare program allowing for voluntarily involving the reproductive cells of another individual than the parents to conceive a child at the discretion of a parent population. #Evolution #Naturalselection #Eugenics #Genetics #ClassicalAndDevelopmentalGenetics #ConservationAndEcologicalGenetics #EvolutionaryAndPopulationGenetics #GeneticEngineering #GeneticsOfPlantsAndAnimalsInAgronomy #Genomics #Peertechz #PeertechzPublications #HumanAndMedicalGenetics #QuantitativeGenetics #PopulationGenetics #PsychiatricGenetics #MicrobialGenetics #MolecularGenetics #GeneCharacterization #Polymorphisms #ControlOfGeneExpression #Epigenetics #TechnicalTipsAndImprovement #EpigeneticsAndChromosomeBiology #FunctionalGenetics #HumanPopulationGenetics #PlantPopulationGenetics #AnimalPopulationGenetics #ComplexTraits #QuantitativeGenetics #StatisticalAndComputationalGenetics #SomaticGeneTherapy #VectorsInGeneTherapy #Viruses #GermlineGeneTherapy #MajorDevelopmentsInGeneTherapy #NonViralMethods #PreventiveGeneTherapy
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