Epileptic Disorders’ Post

Excellent summary on EEG- artifacts and benign variants

How does redox imbalance in astrocytes influence aging of the central nervous system (CNS)? Tsesmelis et al. tried to answer this question by doing research with a conditional astrocyte-specific SOD2-deficient mouse model (SOD2ako). Their research revealed the critical role played by ROS homeostasis in astrocytes in CNS aging. Discover more by clicking here: https://lnkd.in/gwPMUHBp

Our new article about the relationship between frontal alpha asymmetry and behavioural and brain activity indices of reactive inhibitory control has just been published, congrats to all co-authors. https://lnkd.in/dudMKry4

Localization and Lateralization of Ictal Seizure Semiology Source:  A., Leslie, and Jerry J. ‘The Classification of Seizures and Epilepsy Syndromes’. Novel Aspects on Epilepsy, InTech, 12 Oct. 2011. Crossref, doi:10.5772/17860.

Time for #FunFactFriday! 🧠 Let's learn some of the facts about #Epilepsy. Generalized epilepsy can be categorized by motor onset and non-motor onset features. Generalized epilepsy with motor onset features including tonic-clonic, tonic, atonic, myoclonic, myoclonic-atonic, and epileptic spasms. The motor onset features described the dominant feature of the seizure at the beginning. Non-motor onset generalized seizures are more difficult to recognize clinically and are often referred to as absence seizures. Question: What is not a variant of generalized non-motor absence seizure? Choose the correct answer on our website. ⬇

Here is our recent report documenting a remarkable metabolic shift in the human hippocampus affected by temporal lobe epilepsy. This finding may indicate that the TLE hippocampus entails an active metabolic pool producing several anionic phospholipids at the expense of cationinc phospholipids, owing to the dysregulation of the Kennedy and CDP-DG pathways. Click the following link to know more about this exciting work. https://lnkd.in/ddU_g6Z2

Time for #FunFactFriday! Last week we learned how epilepsy is defined and classified. Let's continue this week into the subcategories. Focal onset seizures can be subcategorized into aware and impaired awareness (describing the person’s level of consciousness) then further divided into motor or non-motor (describing clinical manifestations or semiology) during an event. The onset features can allow identification of the area of the brain, lobe or hemisphere that is involved in seizure onset and propagation. Focal seizures can spread widely in the brain to engage bilateral networks, including cortical and subcortical structures, resulting in a tonic-clonic seizure with loss of consciousness. This seizure type is known as a focal to bilateral tonic clonic seizure. Question: Focal non-motor seizures can have clinical manifestation from which of the following categories used as seizure descriptors? You have 5️⃣ answer choices this time. Answer on our website. https://lnkd.in/eemAWdkN

Pivotal role of ACC pyramidal neurons in developing chronic-pain-induced insomnia https://lnkd.in/gax-AJqV

Today's Topic  Epilepsy Or Syncope Unlocking the Enigma: Epilepsy vs. Syncope Demystified with Dr. Mukesh Kumar Every blackout isn't just another seizure episode. When you experience unconsciousness, there are two primary culprits at play: Abnormal electrical activity Disrupted blood flow supply When abnormal electrical activity takes the lead, it's labeled as syncope. Let's unravel the mystery of unconsciousness, understanding the intriguing differences between epilepsy and syncope. Visit - https://lnkd.in/dMhJNfY #EpilepsyAwareness #SyncopeAwareness #NeurologyInsights #HealthEducation #MedicalMystery #DrMukeshKumar #NeurologistDelhi #BrainHealth #MedicalScience #HealthcareExpertise #bestneurologistindelhi

A recent study sought to detect changes in basal ganglia oscillatory activity during re-emergent tremor when attenuated by movement. Results did not indicate a direct role of the basal ganglia circuit on Parkinsonian tremor during such moments. https://loom.ly/SgUjmb4