Peptostreptococcaceae: Difference between revisions
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Microbiome studies of animal feces have corroborated this. Notably, an unclassified group of Peptostreptococcaceae has been reported making up a significant portion of the microbial community in [[domestic cat]]s,<ref name=Bermingham2018>{{cite journal | vauthors = Bermingham EN, Young W, Butowski CF, Moon CD, Maclean PH, Rosendale D, Cave NJ, Thomas DG | display-authors = 6 | title = The Fecal Microbiota in the Domestic Cat (<i>Felis catus</i>) Is Influenced by Interactions Between Age and Diet; A Five Year Longitudinal Study | journal = Frontiers in Microbiology | volume = 9 | pages = 1231 | date = 2018 | pmid = 29971046 | pmc = 6018416 | doi = 10.3389/fmicb.2018.01231 | doi-access = free }}</ref> while other studies have not found a significant presence of Peptostreptococcaceae.<ref name=Bermingham2018/> |
Microbiome studies of animal feces have corroborated this. Notably, an unclassified group of Peptostreptococcaceae has been reported making up a significant portion of the microbial community in [[domestic cat]]s,<ref name=Bermingham2018>{{cite journal | vauthors = Bermingham EN, Young W, Butowski CF, Moon CD, Maclean PH, Rosendale D, Cave NJ, Thomas DG | display-authors = 6 | title = The Fecal Microbiota in the Domestic Cat (<i>Felis catus</i>) Is Influenced by Interactions Between Age and Diet; A Five Year Longitudinal Study | journal = Frontiers in Microbiology | volume = 9 | pages = 1231 | date = 2018 | pmid = 29971046 | pmc = 6018416 | doi = 10.3389/fmicb.2018.01231 | doi-access = free }}</ref> while other studies have not found a significant presence of Peptostreptococcaceae.<ref name=Bermingham2018/> |
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Peptostreptococcaceae have been of interest for several other bowel diseases as biological marker or causative agent. Decreased abundance has been reported for [[Crohn's disease]],<ref name=Pascal2017>{{cite journal | vauthors = Pascal V, Pozuelo M, Borruel N, Casellas F, Campos D, Santiago A, Martinez X, Varela E, Sarrabayrouse G, Machiels K, Vermeire S, Sokol H, Guarner F, Manichanh C | display-authors = 6 | title = A microbial signature for Crohn's disease | journal = Gut | volume = 66 | issue = 5 | pages = 813–822 | date = May 2017 | pmid = 28179361 | pmc = 5531220 | doi = 10.1136/gutjnl-2016-313235 }}</ref> while the genus ''[[Peptostreptococcus]]'' appears to be more common in patients diagnosed with [[colorectal cancer]].<ref name=Ahn2013>{{cite journal | vauthors = Ahn J, Sinha R, Pei Z, Dominianni C, Wu J, Shi J, Goedert JJ, Hayes RB, Yang L | display-authors = 6 | title = Human gut microbiome and risk for colorectal cancer | journal = Journal of the National Cancer Institute | volume = 105 | issue = 24 | pages = 1907–1911 | date = December 2013 | pmid = 24316595 | pmc = 3866154 | doi = 10.1093/jnci/djt300 }}</ref> |
''[[Clostridioides difficile]]'' is a notable human pathogen in this family. Peptostreptococcaceae have been of interest for several other bowel diseases as biological marker or causative agent. Decreased abundance has been reported for [[Crohn's disease]],<ref name=Pascal2017>{{cite journal | vauthors = Pascal V, Pozuelo M, Borruel N, Casellas F, Campos D, Santiago A, Martinez X, Varela E, Sarrabayrouse G, Machiels K, Vermeire S, Sokol H, Guarner F, Manichanh C | display-authors = 6 | title = A microbial signature for Crohn's disease | journal = Gut | volume = 66 | issue = 5 | pages = 813–822 | date = May 2017 | pmid = 28179361 | pmc = 5531220 | doi = 10.1136/gutjnl-2016-313235 }}</ref> while the genus ''[[Peptostreptococcus]]'' appears to be more common in patients diagnosed with [[colorectal cancer]].<ref name=Ahn2013>{{cite journal | vauthors = Ahn J, Sinha R, Pei Z, Dominianni C, Wu J, Shi J, Goedert JJ, Hayes RB, Yang L | display-authors = 6 | title = Human gut microbiome and risk for colorectal cancer | journal = Journal of the National Cancer Institute | volume = 105 | issue = 24 | pages = 1907–1911 | date = December 2013 | pmid = 24316595 | pmc = 3866154 | doi = 10.1093/jnci/djt300 }}</ref> |
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== References == |
== References == |
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Revision as of 17:45, 24 August 2023
| Peptostreptococcaceae | |
|---|---|
Scientific classification 
| |
| Domain: | Bacteria |
| Phylum: | Bacillota |
| Class: | Clostridia |
| Order: | Eubacteriales |
| Family: | Peptostreptococcaceae Ezaki 2010[1] |
| Genera | |
|
Acetoanaerobium | |
The Peptostreptococcaceae are a family of Gram-positive bacteria in the class Clostridia.
Several members of the Peptostreptococcaceae are well known inhabitants of the digestive tract. Microbiome studies of animal feces have corroborated this. Notably, an unclassified group of Peptostreptococcaceae has been reported making up a significant portion of the microbial community in domestic cats,[2] while other studies have not found a significant presence of Peptostreptococcaceae.[2]
Clostridioides difficile is a notable human pathogen in this family. Peptostreptococcaceae have been of interest for several other bowel diseases as biological marker or causative agent. Decreased abundance has been reported for Crohn's disease,[3] while the genus Peptostreptococcus appears to be more common in patients diagnosed with colorectal cancer.[4]
References
- ^ Page Family Peptostreptococcaceae on "LPSN - List of Prokaryotic names with Standing in Nomenclature". Deutsche Sammlung von Mikroorganismen und Zellkulturen. Retrieved 2022-11-08.
- ^ a b Bermingham EN, Young W, Butowski CF, Moon CD, Maclean PH, Rosendale D, et al. (2018). "The Fecal Microbiota in the Domestic Cat (Felis catus) Is Influenced by Interactions Between Age and Diet; A Five Year Longitudinal Study". Frontiers in Microbiology. 9: 1231. doi:10.3389/fmicb.2018.01231. PMC 6018416. PMID 29971046.
- ^ Pascal V, Pozuelo M, Borruel N, Casellas F, Campos D, Santiago A, et al. (May 2017). "A microbial signature for Crohn's disease". Gut. 66 (5): 813–822. doi:10.1136/gutjnl-2016-313235. PMC 5531220. PMID 28179361.
- ^ Ahn J, Sinha R, Pei Z, Dominianni C, Wu J, Shi J, et al. (December 2013). "Human gut microbiome and risk for colorectal cancer". Journal of the National Cancer Institute. 105 (24): 1907–1911. doi:10.1093/jnci/djt300. PMC 3866154. PMID 24316595.
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